Active Research Projects
About us
Our laboratory has elucidated the contribution of rare de novo and inherited mutations to the genetic risk of schizophrenia and develops relevant model systems to understand their impact on neural mechanisms and advance neuropsychiatric therapeutics.
News
October 24, 2019
Schizophrenia risk gene linked to cognitive deficits in mice
Restoring “master regulator” reverses behavioral, neuronal abnormalities.
October 09, 2019
Columbia Scientists Reverse Core Symptom of Schizophrenia in Adult Mice
Findings reveal potential new strategy for treating people diagnosed with schizophrenia.
July 27, 2018
New Uses for Old Medications
Even drugs whose development was stalled or canceled might show promise for illnesses they were never meant to treat.
Photos
Contralateral axon-terminal branching of EGFP-labeled neurons from coronal sections of P56 (8 weeks) brains of mice in utero electroporated at E15.5. green: EGFP, red: NeuN.
EGFP-labeled neurons migration at E17.5 with in Utero electroporation at E14.5 mouse brain.
Ipsilateral axon branching of EGFP-labeled neurons from coronal sections of P8.5 brains of mice in utero electroporated at E15.5. green: EGFP, red: NeuN.
